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14th October, 2009
European Survey on Standards of Care in paediatric oncology centres. J. R. Kowalczyk, M. Samardakiewicz, K. Pritchard-Jones, R. Ladenstein, S. Essiaf, E. Fitzgerald, G. Petrarulo, G. Vassal - EJC , Volume 61, pp 11-19
8th December, 2014
The purpose of this website is to provide good and clear information about the long-term effects of childhood cancer and to refer to the various late-effects outpatient clinics (LATER outpatient clinics) that exist in the Netherlands.
8th September, 2015
Cancer in young people is rare, but it is still a major health issue in Europe. Each year, more than 6,000 young people in Europe die of cancer. There are more than 300,000 European childhood cancer survivors (in 2020, they will be nearly half a million): two-thirds of them have some late side effects of treatment, that are severe and impact on the daily life of half of those affected.
Oncogenes are genes that can potentially cause cancer. When they are normal, they help to regulate cell growth and division. However, when they are altered or mutated, they can lead to abnormal cell growth and proliferation, eventually fostering the development of cancer.
As we delve into twelve centuries of scientific advancement, the intricate mechanisms that govern human life continue to unravel before our eyes. One encapsulating beacon of this evolution lies within the realm of genetics. Understanding genetic terminology such as Oncogenes is crucial not just for scientists and healthcare practitioners, but also for anyone interested in human health, disease progression, and potential treatment methods.
Oncogenes are a fascinating subcategory of genes that, when altered or mutated, have the potential to cause normal cells to turn cancerous. Unlike other genes, whose primary function is to control cell growth and division, Oncogenes can disrupt this orderly process. This is primarily due to the deregulation of the cell multiplication process that occurs during Oncogenes mutation.
The complex nature of Oncogenes and their role are of vast significance. Their study opens vistas to understanding the core pathways that lead to the development and progression of various cancers.
Naturally, every Oncogene starts as a regular gene, also known as a proto-oncogene. Several factors such as radiation exposure, chemical carcinogens, and certain viruses can cause these genes to mutate, thus converting them into Oncogenes. The constant evolution and significant role of mutations in Oncogenes cannot be undermined, especially when considering their impact on cancer development.
At a fundamental level, Oncogenes contribute to cancer by disrupting normal cell behavior. This disruption allows cells to multiply in an uncontrolled manner, thereby leading to the formation of a tumor. Oncogenes can contribute to various types of cancer, such as lung, breast, and colorectal cancer.
Detecting the presence of Oncogenes is imperative for diagnosing disorders associated with them. Typically, diagnostic procedures involve examining tissue or blood samples to identify gene mutations. Technological advancements have made it possible to screen for these mutations, thereby providing promising directions for early disease detection and treatment.
Decoding the mysteries surrounding Oncogenes has opened up various treatment options. Strategies such as targeted therapies aim to attack specific cancer cells, thus minimizing the impact on healthy cells. Moreover, the role of gene therapy in managing disorders linked to Oncogenes cannot be dismissed. Through altering, replacing, or supplementing nonfunctional Oncogenes, gene therapy provides a promising pathway for effective disease management.
Unraveling the mysteries surrounding Oncogenes offers an insight into how cancers develop and progress. Understanding their function, diagnosing their presence, and treating diseases resulting from Oncogenes mutations represent critical junctures in modern healthcare. The intriguing world of Oncogenes undoubtedly holds considerable potential to revolutionize healthcare in the future.
Oncogenes are essentially normal genes turned bad. Unlike typical genes, which help control cell growth and division, Oncogenes harbor mutations that can cause cells to multiply out of control, potentially leading to cancer.
Several factors, including exposure to certain chemicals, radiation, and certain viruses, can trigger the mutation of regular genes to Oncogenes.
Oncogenes contribute to cancer by disrupting the normal cell process, which can cause cells to multiply uncontrollably and form a tumor.
Oncogenes can contribute to various types of cancers, including but not limited to lung, breast, and colorectal cancer.
Detection techniques typically involve examining tissue or blood samples to identify genetic mutations, thus confirming the existence of Oncogenes.
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